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In this video, we delve into the vital topic of enhancing pleasure in sexual relationships. Sexual pleasure is not just a physical experience; it fosters emotional connections and strengthens bonds between partners. By prioritizing pleasure, we can create more satisfying and harmonious relationships that lead to improved communication and deeper intimacy. We outline 10 effective strategies designed to enhance our sexual experiences, from improving communication about desires to exploring new techniques. Engaging in open dialogue outside the bedroom creates a safe space for discussing preferences, ultimately enriching our connection. Together, let's embark on this journey towards greater intimacy! #SexualHealth #IntimacyMatters Acelere O Metabolismo A Testo Vem Shorts Testosterone Male

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Today we are going to talk about the main medicines that can cause, erectile dysfunction. There are really key things you should know. the key with erectile dysfunction is drugs that affect the nervous system, and some that lower testosterone levels, or inhibit testosterone action can cause ED. It is estimated that 25 percent of cases of erectile dysfunction are because of medications and may be reversible by decreasing dose or changing the medicine. Medication induced erectile dysfunction is widely prevalent and is caused by medications from a variety of medical fields. Finasteride and dutasteride had the highest reported causes of erectile dysfunction. Finasteride and dutasteride work by irreversibly binding to an enzyme 5-α reductase, thereby preventing the conversion of testosterone to dihydrotestosterone which the more potent form of testosterone. Finasteride binds to type II 5 α reductase which is found mostly in the prostate and genital tract. Dutasteride binds to both type 1 (found mostly in extraprostatic tissues) and type II 5 α reductase. dihydrotestosterone is vital for physiologic erections as it activates nitric oxide synthase and increases blood flow in cavernosal tissue. patients on finasteride are more likely to have a loss of libido, ejaculatory disorder, and erectile dysfunction. Neuropsychiatric medications accounted for the second common cause of erectile dysfunction. The antipsychotic paliperidone and the selective serotonin reuptake inhibitors citalopram and sertraline were found to have elevated high rate of erectile dysfunction. In addition, there are many neuropsychiatric medications with high frequencies of erectile dysfunction including escitalopram, quetiapine, olanzapine, fluoxetine, venlafaxine, risperidone, aripiprazole, gabapentin, pregabalin, and oxycodone. Neuropsychiatric medication induced erectile dysfunction is a well described phenomenon. Neuropsychiatric medications have a variety of adverse effects due to the multiple receptors targeted throughout the body. These receptors include central and peripheral serotonergic, adrenergic, dopaminergic, cholinergic, histaminergic, and melanocortin receptors. Antipsychotics inhibit dopamine receptors with varying potency, which results in both direct and indirect inhibition of erections. Dopamine is known to play an important role in emotional sexual behavior and may also directly facilitate erections. Antipsychotics can cause hyperprolactinemia by inhibiting D2 receptors in the tuberoinfundibular system. This leads to decreased gonadotropin production and secondary hypogonadism. The different antipsychotics affect various receptors and this may have varying side effects. For example, the antipsychotics that block α1 adrenergic receptors may cause retrograde ejaculation but not erectile dysfunction. Benzodiazepines may cause sexual dysfunction from potentiation of GABA in the reticular and limbic system and by affecting the serotonin and dopaminergic pathways. Cardiological medications accounted some of rug induced erectile dysfunction. Beta blockers have been shown to be associated with ED, likely secondary to suppression of central nervous system sympathetic outflow. Non–cardioselective beta antagonists like propranolol have a higher incidence of erectile dysfunction than cardioselective beta antagonists which avoid beta 2 inhibition resulting in vasoconstriction of the corpora cavernosa. Nebivolol has the greatest selectivity for beta 1 receptors as well as endothelial nitric oxide vasodilatory effects, and has been shown to have a positive effect on erections. thiazide diuretics can also induce erectile dysfunction. In contrast, there is less evidence that loop diuretics such as furosemide cause ED, which should be considered by physicians treating hypertensive men. Some data suggests that ejaculatory dysfunction with calcium channel blockers (from decreased bulbocavernosus muscle force). Angiotensin-converting enzyme inhibitors and angiotensin receptor blockers are common antihypertensives that may actually improve erectile function. Angiotensin converting enzyme inhibitors prevent the formation of angiotensin II while angiotensin receptor blockers block the peripheral vasoconstriction caused by angiotensin II. Without angiotensin IIs effects, there is less penile vasoconstriction and collagen remodeling of the corpora cavernosa. In most studies, ACE inhibitors and ARBs have not been associated with ED and some studies even showed a beneficial effect. Medicines used for Dermatology and Immunology can also cause erectile dysfunction. Isotretinoin is a sebum suppressive medication used for acne which can also lead to erectile tretinoin. It is possible that acne is related to depression, which is a known risk factor for ED. Adalimumab can also cause ED. Adalimumab is a tumor necrosis factor (TNF) α blocker.

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Watch the original video titled WARNING: The Negative Effects of Masturbation: 👉https://www.youtube.com/watch?v=E_5xl0w_mXE Masturbation & Testosterone Levels - Truth & Clinical Facts 👉https://www.youtube.com/watch?v=o1kxBpCE3YA How To Stop Masturbating & Should You? 👉https://www.youtube.com/watch?v=TK5oY6aKmvA #drsamrobbins #masturbation ==================================== 🍌 Warning: Masturbation Shrinks Your Penis ==================================== So today, I’m going to talk again about masturbation. It’s interesting how many questions I get asked about this topic. However, as I’ve stated before, this channel is for YOU and I discuss topics that you’re most interested in and have questions about. So with that said, let’s discuss today’s question: Dr. Sam - I heard and read that masturbation can shrink your penis, is this true? I masturbate about 3-5x daily, sometimes as often as 7x daily. I’m worried. I don’t want my penis to shrink. Hell, I want it to get bigger. Anyway, what’s your thought? Growing up, I never thought that masturbation would shrink my penis. I mean, this was never even talked about with my buddies. But these days, I get asked this question a ton of times for some reason. So, here’s the truth - YES, masturbation can cause your penis to shrink. Actually, let me rephrase that, excessive masturbation can cause shrinkage. Of course, the next question is, what’s “excessive”? Well, 4-7x daily is a lot. In fact, if you’re masturbating that often, daily, every day - you need to get a job or something because you’ve got way too much time on your hands. And I’ve spoken about the “Negative Effects of Masturbation” in another video, which I’ll give you the direct link to that at the end of this video. Masturbation Is Normal and Healthy, But... Let’s get something straight before I continue - Masturbating is normal and healthy. Men and women do it. You’ve seen dogs hump your leg or a toy, they do it too. And, when you’re younger and have higher testosterone and optimal hormone levels, you’re hornier and will masturbate or have sex, more often. And with that said, I think 1-2x daily is plenty. What’s Excessive Masturbation? Now, another question you may be asking is “why or how does excessive masturbating cause penis shrinkage”? Well, it’s very similar to working out. Even though your penis is an organ consisting of spongy tissue, it still contains muscle tissue - more specifically, smooth muscle. Not the same as your bicep or chest muscle, which is called skeletal muscle. Needless to say, and even though it’s not “clinically proven”, you can most certainly grow your penis and at the same time, shrink it. TRUST ME ON THIS, growing your penis is very possible and it happens plenty of times. It’s been shown with lots of guys. And don’t even bother posting something below in the comments section arguing otherwise. Now, I’m not going to talk about penis GROWTH today. However, excessive masturbating call lower testosterone levels and create hormonal imbalances, which can cause penis shrinkage. If you masturbate or have sex 1-2 daily, it’s fine. But if you do it many times daily and thus, “excessively”, then you’ll have hormonal imbalances many times during the day, all of which, over time, can cause penis shrinkage. Additionally, just as LACK of use will cause shrinkage, so will OVER use. Just like going to the gym - you don’t want to under or over train, if your goal is maximum muscle growth. Remember, everything in the body requires stimulation and recuperation, if you want progress. Even your brain - you need to learn something new daily, read more, and so forth to continue to build new brain cells, synapsis and so forth. That’s the stimulation. While at the same time, you need proper sleep, healthy fats, vitamins and minerals to help the brain recuperate and thus, remember what was learned to prevent memory and cognitive problems. Yes, I’m simplifying it all - but I hope you get the point. So, stimulate your penis. Get erections. Do Kegel exercises. Have sex or masturbate. Have orgasms. Then, let it rest and recuperate. I guess like anything life, moderation is the key. Lastly, you may be wondering “well, how much will it shrink if I masturbate excessively”? I have no idea. Anecdotally, it’s not much. More girth than length. Maybe half an inch or so, maybe more if you’ve got more? But it’s not just about the shrinkage, it’s about having optimal hormone levels so you perform at your best, now and in the future. It’s about penis and prostate health. ======================================== Thank you for watching. Please feel free to comment, like or share with your friends. Subscribe to Dr.Sam Robbins's official Youtube channel http://drsam.co/yt/subscribe Like us on Facebook https://www.facebook.com/DrSamRobbins ======================================== Thanks DrSamRobbins

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